Check out Onnit's MCT
https://www.onnit.com/onnit-mct-oil/
I use their emulsified version in my coffee. Love it.
Thanks for the reply.
Looks like that product is 30% C12 (lauric acid), 31% C10 (capryic acid), and 38% C8 (caprylic acid). On a good note, all three of those MCT's are saturated fatty acids plus that product is 100% coconut oil (no palm oil).
Now it's just a matter of determining:
1. Will this MCT oil work better for extraction than standard coconut oil?
I think it will because it should be 100% saturated fatty acids vs. coconut oil's roughly 93%.
2. Will this MCT oil work better for oral ingestion than standard coconut oil?
I think it will because it's 100% medium chains (vs. 63% in coconut oil) and it's my understanding that medium chains are absorbed and transported directly to the liver where the thc is converted into 11 hydroxy thc, which is supposed to be up to 3x more powerful. However, I've seen other claims that only the medium chain C12 (lauric) is processed by the liver.....(see "where stuff gets weird" below). I've also read that C12 (lauric) is 66% processed by the liver and 34% processed by the lymphatic system.
That leads to the next question though.... Choosing the best type of 100% Coconut oil based MCT oil.
Choosing between options like 100% C6, 100% C8, 100% C10, 100% C12, or a mixed MCT. If mixed, it's a matter of deciding if the shorter C's should be favored over the longer C's (and if Lauric Acid, C12 should be avoided completely).
Here is where stuff gets weird....
I've seen websites claiming that C12 (lauric) acts more like a long chain triglyceride than a medium chain one, despite it being classified as a medium chain triglyceride. And again, I've seen websites claim that MCT's are processed by the liver more so than LCTs and therefore produce more 11 hydroxy thc. But I've seen other sites claiming that the shorter MCT's (namely 8 and 10) are actually less likely to go through the liver pathway than C12 (lauric), because they can be converted into ketones and used in the brain instead. My suspicion is that they are wrong and that both C8 and C10 do indeed get processed by the liver first, prior to either being stored as fat or being converted into ketones to feed the brain. These same sites report that C12 is the technically classified MCT that HAS to be processed by the liver. See link below.
https://blog.bulletproof.com/what-is-mct-oil-vs-coconut-oil/
"
C8, Caprylic Acid (Brain Octane)- The rarest ~6% of coconut oil:
It has potent anti-microbial properties (way more potent than lauric acid) to help you maintain a healthy gut, and it is the fastest to metabolize in the brain. (This is
Brain Octane Oil.) Your liver does not need to process this rare type of MCT, and it only takes 3 steps for your body to turn it into ATP, the cellular fuel you use.[5] Sugar takes 26 steps. This is why Brain Octane is so good at suppressing cravings and is the most powerful oil to put in Bulletproof Coffee. You would need 18 tablespoons of coconut oil to get just one tablespoon of Brain Octane.
C10, Capric Acid – Around ~9% of coconut oil:
This is the second shortest form of MCT, also rare. It is slower to turn into energy but more affordable than C8.
XCT Oil is triple-distilled in a non-oxygen atmosphere with no solvents ever used, and it contains C10 and C8, because these are the only two MCT oils that turn into ATP quickly without the liver. You would need 6 tablespoons of coconut oil to get one tablespoon of XCT oil.
C12, Lauric Acid: Around ~50+% of coconut oil:
It requires a pit stop in the liver rather than getting immediately converted into energy like the other MCTs above."
So I guess what I really need to know is which MCT (8, 10, or 12) is/are most likely to be processed by the liver?
Products. like the one you linked to, seem to have the same % of C12 as coconut oil, but they gain more C8 and C10 in exchange for less LCT's and less Unsaturated fats. Since they have the same amount of C12 and overall more saturated fat than standard coconut oil, I'd say they have to be better for the extraction of thc. It's just a question of whether or not the additional C8 and C10 will be processed by the liver or not. I guess it's also a question of whether the increased rapidness of absorption that C8 and C10 offer will substantially offset the reduction of 11 hydroxy thc production they might induce by not passing through the liver....